The life of a crested newt defies explanation from the very start.
Why such a curse persists in every species of crested newt has long been a total mystery. In fact, for more than 150 years after Rusconi's initial observation, no scientist could figure out why so many newt embryos were doomed from the very start.for researchers Herbert Macgregor and Heather Horne to make a pivotal discovery, learning the 'cursed' newt embryos were dying from a weird genetic mutation in their number 1 chromosome, which made it longer.
Surprizingly, 1B isn't exactly perfect either, with a different 'mistake' that renders critical information useless.If a newt egg is fertilized by a sperm cell with the same variation of chromosome 1 , it results in a deadly combination of 1A+1A or 1B+1B in the offspring. The other essential half is missing.
This isn't unusual in itself. Nature has a neat way of minimizing the odds of an embryo inheriting broken genes such as these, shuffling sections of chromosomes from each parent around with one another.in an article from 2021,"chromosomes can swap the faulty DNA for functional DNA." But in crested newts, chromosome 1 pairs aren't shuffled with one other, in spite of other pairs of chromosomes receiving a good mix-up. This means those with a deadly chromosome 1 pairing barely make it off the developmental starting block.
Those embryos that do survive will carry one 1A and one 1B chromosome, meaning that the next generation is doomed to undergo the exact same losses, over and over.
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