Molecular insights point to neuronal underpinnings of obesity. Scientists studying the nucleus accumbens in rats found differences between obesity-prone and obesity-resistant animals, with the former showing delayed glucose entry and excess glutamate levels. These findings suggest a defect in neuro
Researchers are investigating the role of the brain’s nucleus accumbens in driving overeating and obesity. A recent study using rat models found differences in the nucleus accumbens between obesity-prone and obesity-resistant rats, showing that glucose took longer to enter the nucleus accumbens in obesity-prone animals. Furthermore, excess levels of glutamate, an excitatory neurotransmitter, were found in these rats, suggesting a defect in a neurotransmitter recycling process.
“These brain motivation centers evolved to help us survive; finding food and having sex are essential to the survival of an individual and of a,” said Carrie Ferrario, Ph.D., associate professor in the Department of Pharmacology at the University of Michigan Medical School. Previous research from Ferrario’s lab pinpointed differences in the nucleus accumbens in obesity-prone and obesity-resistant rats. Their latest study, published in the, tracked what was happening in real-time in the brain when these animals were presented with glucose, a type of sugar, labeled with a tracer. The tracer allowed the researchers to measure this new sugar in the brain.
They found that glucose was taking longer to get into the nucleus accumbens of obesity-prone animals. This balance, and therefore brain activity, is different in obesity-prone vs. obesity-resistant rats.
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